Selective persistence of HIV-1-infected T cell clones can occur through immune reprogramming driven by defective, transcriptionally active proviruses

Martin V. Hamann, Lisa Brauckmann, Christoph Schwarz, Michael Spohn, Ramon Stoeck, Sabrina M. Leddy, Julie Frouard, Maisha Adiba, Riekje Winzer, Fabian Hausmann, Sanamjeet Virdi, Adam Grundhoff, Cedric Feschotte, Daniel Sauter, Frauke Muecksch, Nadia R. Roan, Eva Tolosa, Ulrike C. Lange

DOI: 10.1371/journal.ppat.1014618

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